Patrick R. H. Steinmetz

The paper shows that starvation-induced cell loss in sea anemones occurs via epithelial cell extrusion, with ERK1/2 signaling regulating extrusion density.

This study shows that the RNA labeling compound EU integrates into DNA in some animal species, raising concerns about its specificity for RNA labeling.

This study shows that starvation induces reversible G1/G0 arrest in Vasa2+/Piwi1+ cells of Nematostella vectensis, with refeeding-dependent TOR signaling controlling cell cycle re-entry.

The paper identifies and characterizes a multipotent stem-like cell population in Nematostella vectensis that generates both germline and somatic cells, including neurons.

The paper studies how feeding and starvation affect body size and cellular processes in sea anemones, finding that cell cycle shifts and growth rates are nutritionally regulated.

The paper identifies NvPrdm14d-expressing endodermal progenitor cells in Nematostella vectensis that contribute to non-ectodermal neurogenesis, revealing new insights into the molecular regulation of this process.

The study identifies gene duplications and co-option of effector modules as mechanisms driving muscle cell type diversification in sea anemones.