Darrin H. Brager

The paper shows that cortical spiking variability arises from weak synchrony rather than intrinsic properties, with synchrony dynamics shifting in response to sensory input.

This study shows oxytocin increases excitability and synaptic transmission in dentate gyrus granule cells by altering membrane properties and presynaptic release probability.

This study identifies impaired thalamic burst firing in a mouse model of Fragile X syndrome, linking it to altered voltage sensitivity of Ca2+-dependent low threshold spikes.

The paper examines CA2 neuronal responses to social stimuli in a rat model of Fragile X Syndrome and finds impaired responses despite normal social behaviors.

This study investigates intrinsic excitability changes in hippocampal inhibitory interneurons in Fragile X mice, finding increased Ih in a specific subpopulation.

This study shows that miR-324-5p regulates dendritic spine structure and hippocampal long-term potentiation in mice by affecting synaptic proteins and plasticity.

The paper investigates dendritic spike generation in prefrontal cortex neurons of Fragile X mice and finds impaired sodium channel function leading to altered input-output transformation.

This study shows that impaired A-type potassium channel function in FXS mice leads to reduced dendritic spike initiation and TA-LTP, suggesting a potential therapeutic target for cognitive impairments.

The paper identifies two distinct subtypes of low threshold interneurons in mouse CA1 stratum oriens based on spike frequency adaptation and Ih current density.

This study shows that FMRP regulates D-type K+ channels in ET neurons of the prefrontal cortex, affecting action potential threshold in FX mice.