Author
Kartik Pattabiraman
Yale UniversityORCID 0000-0002-3924-426XUnclaimed ORCID profile · follow to get notified of new preprints
Biology Disciplines
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2019-2026 · peak 2/yrCo-Author Collaboration Network
10 top collaborators · 43 mutual co-author links6 papers
Recent·Most cited
- NeuroscienceNaturePeer-reviewed
Competing Programs Shape Cortical Sensorimotor-Association Axis Development
Jeremiah Tsyporin, Menglei Zhang, Cai Qi…·Nenad Šestan
The paper presents the MIND model, showing that competing induction and exclusion programs shape the sensorimotor-to-association axis in the neocortex across species.
- NeurosciencebioRxivPreprint
Molecular Specification of Claustro-Amygdalar and Paleocortical Neurons and Connectivity
Navjot Kaur, Rothem Kovner, Forrest O. Gulden…·Nenad Šestan
This study identifies SOX4, SOX11, and TFAP2D as critical regulators of ventropallial excitatory neuron development and PFC connectivity in mice, with conserved roles in humans.
- NeuroscienceProceedings of the National Academy of SciencesPeer-reviewed
Transcriptional Network Orchestrating Regional Patterning of Cortical Progenitors
Athéna Rebecca Ypsilanti, Kartik Pattabiraman, Rinaldo Catta-Preta…·John L.R. Rubenstein
The paper identifies a transcription factor network and enhancers involved in cortical regional patterning using mouse embryonic data.
- NeurosciencebioRxivPreprint
Regulation of Prefrontal Patterning, Connectivity and Synaptogenesis by Retinoic Acid
Mikihito Shibata, Kartik Pattabiraman, Belén Lorente-Galdós…·Nenad Šestan
This study identifies retinoic acid signaling as critical for prefrontal cortex development and connectivity in primates, showing its role in molecular patterning, synaptogenesis, and evolutionary expansion.
- NeurosciencebioRxivPreprint
Hominini-Specific Regulation of CBLN2 Increases Prefrontal Synaptogenesis
Mikihito Shibata, Kartik Pattabiraman, Sydney Keaton Muchnik, Nenad Šestan
The paper identifies Hominini-specific regulation of CBLN2 that drives increased synaptogenesis in the human prefrontal cortex, linking it to neuropsychiatric disorder risk.