Author
Mikihito Shibata
Yale UniversityORCID 0009-0008-5588-4583Unclaimed ORCID profile · follow to get notified of new preprints
Biology Disciplines
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2019-2026 · peak 2/yrCo-Author Collaboration Network
10 top collaborators · 43 mutual co-author links6 papers
Recent·Most cited
- NeuroscienceNaturePeer-reviewed
Competing Programs Shape Cortical Sensorimotor-Association Axis Development
Jeremiah Tsyporin, Menglei Zhang, Cai Qi…·Nenad Šestan
The paper presents the MIND model, showing that competing induction and exclusion programs shape the sensorimotor-to-association axis in the neocortex across species.
- NeurosciencebioRxivPreprint
Adaptive Evolution of Gene Regulatory Networks in Mammalian Neocortical Neurons
Zhuo Li, Navjot Kaur, Gabriel Santpere…·Nenad Šestan
The paper identifies mammalian-specific gene regulatory elements and their role in neocortical neuron subtype diversity and connectivity, linking them to neurodevelopmental disorders.
1 citation - NeuroscienceSciencePeer-reviewed
Molecular programs of regional specification and neural stem cell fate progression in developing macaque telencephalon
Nicola Micali, Shaojie Ma, Mingfeng Li…·PASKO T. RAKIC
The paper uses single-cell transcriptomics to identify molecular programs in developing macaque telencephalon, revealing primate-specific signaling and gene roles in regional specification and neural stem cell fate.
83 citationstop 1% - NeurosciencebioRxivPreprint
Regulation of Prefrontal Patterning, Connectivity and Synaptogenesis by Retinoic Acid
Mikihito Shibata, Kartik Pattabiraman, Belén Lorente-Galdós…·Nenad Šestan
This study identifies retinoic acid signaling as critical for prefrontal cortex development and connectivity in primates, showing its role in molecular patterning, synaptogenesis, and evolutionary expansion.
- NeurosciencebioRxivPreprint
Hominini-Specific Regulation of CBLN2 Increases Prefrontal Synaptogenesis
Mikihito Shibata, Kartik Pattabiraman, Sydney Keaton Muchnik, Nenad Šestan
The paper identifies Hominini-specific regulation of CBLN2 that drives increased synaptogenesis in the human prefrontal cortex, linking it to neuropsychiatric disorder risk.