Author

Anahid Ehteda

UNSW SydneyORCID 0000-0002-4118-1499Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

Aaminah KhaniD· 1 shared Alvin Lee· 1 shared Anjana GopalakrishnaniD· 1 shared Benjamin S. RayneriD· 1 shared Chelsea MayohiD· 1 shared Chi Kin IpiD· 1 shared Claire Xin SuniD· 1 shared Daisy KavanaghiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

Aaminah KhanORCID

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2 papers

Recent·Most cited

Cancer BiologyScience Translational MedicinePeer-reviewedJan 14, 2026

Chromatin remodeling with combined FACT and BET inhibition disrupts oncogenic transcription in Diffuse Midline Glioma

Holly Holliday, Aaminah Khan, Anahid Ehteda…·David Simon Ziegler

This study shows that combining FACT and BET inhibition disrupts oncogenic transcription and extends survival in Diffuse Midline Glioma models.

6 citationstop 2%

humanbulk RNA-seq

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Cancer BiologybioRxivPreprintSep 21, 2025

Selective degradation of oncogenic extrachromosomal DNA by type I-E CRISPR

Ensieh M. Poursani, Vu Pham, Anahid Ehteda, Orazio Vittorio

This study demonstrates that type I-E CRISPR can selectively degrade oncogenic extrachromosomal DNA in cancer cells, reducing oncogene signaling and tumor proliferation.

1 citation

humangenetic perturbation / CRISPR

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