Author

Stefania D’Apice

University of Naples Federico IIORCID 0000-0002-4151-5153Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Alessandro VitiiD· 1 shared Antonella Fiordelisi· 1 shared Antonietta Buonaiuto· 1 shared Antonio BiancoiD· 1 shared Antonio PezoneiD· 1 shared Antonio Pisani· 1 shared Cinzia PerrinoiD· 1 shared Daniela SorrientoiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperPathology

Alessandro VitiORCID

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1 paper

Recent·Most cited

Early mitophagy defects and impaired mitochondrial energy metabolism drive target organ damage progression: lessons from the Fabry heart

Jessica Gambardella, Antonella Fiordelisi, Federica Andrea Cerasuolo, Antonietta Buonaiuto, Roberta Avvisato, Alessandro Viti, Eduardo Sommella, Pietro Campiglia, Valeria D’Argenio, Nella Prevete, Antonio Pezone, Stefania D’Apice, Giovanna Giuseppina Altobelli, Fahimeh Varzideh, Shivangi Pande, , Guido Iaccarino

This study identifies mitochondrial dysfunction and impaired mitophagy as key drivers of cardiac damage in Fabry disease and shows that L-arginine supplementation can prevent cardiac abnormalities.

Cell Reports · Oct 26, 2026Peer-reviewedPathology