Author

Sébastien G. Bouret

InsermORCID 0000-0002-4174-9769Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 5 Organism:mouse· 5 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:database / resource· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Fabienne SchalleriD· 3 shared Françoise MuscatelliiD· 3 shared Pierre-Yves BarelleiD· 3 shared Catarina Santos· 2 shared Émilie CaroniD· 2 shared Felix Omnes· 2 shared Jessica Klucznik· 2 shared Julien Maillard· 2 shared

Activity by Year

2018-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 32 mutual co-author links

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3 shared papersNeuroscience

Fabienne SchallerORCID

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Inhibition of oxytocin neurons during key periods of development has long-term behavioural and body composition effects.

Fabienne Schaller, Marie-Sophie Alifrangis, Pierre-Yves Barelle, Catarina Santos, Roman Tyzio, Alessandra Bertoni, Felix Omnes, Yasmine Belaïdouni, Francesca Bader, Emilie Pallesi‐Pocachard, Jean‐Luc Gaïarsa, Sébastien G. Bouret, Françoise Muscatelli

This study inhibits oxytocin neurons during different developmental stages in mice, revealing long-term behavioral and metabolic effects depending on timing and sex.

Molecular Autism · Sep 9, 2026Peer-reviewedNeuroscience

Vesicle-mediated oxytocin release drives melanocortin circuit maturation during a neonatal critical period

Pierre-Yves Barelle, Fabienne Schaller, Soyoung Park, Émilie Caron, Jessica Klucznik, Phillipe Ciofi, Françoise Muscatelli, Sébastien G. Bouret

This study shows that oxytocin release during a neonatal critical period is essential for melanocortin circuit development and metabolic function.

bioRxiv · Apr 10, 2025 · PreprintNeuroscience

Investigation of a Novel Mouse Model of Prader-Willi Syndrome with Invalidation of Necdin and Magel2

Pierre-Yves Barelle, Alicia Sicardi, Fabienne Schaller, Julie Buron, Denis Becquet, Felix Omnes, Françoise Watrin A, Catarina Santos, Clément Menuet, Anne‐Marie François‐Bellan, Émilie Caron, Jessica Klucznik, Vincent Prévot, Sébastien G. Bouret, Françoise Muscatelli

This study creates a novel mouse model with deletion of Necdin and Magel2 genes, revealing functional interactions and phenotypes relevant to Prader-Willi Syndrome.

bioRxiv · Jul 24, 2024 · PreprintSystems Biology

Defective Autophagy in Sf1 Neurons Perturbs the Metabolic Response to Fasting and Causes Mitochondrial Dysfunction

Bérengère Coupé, Corinne Leloup, Kwame Asiedu, Julien Maillard, Luc Pénicaud, Tamás L. Horváth, Sébastien G. Bouret

This study shows that autophagy in Sf1 neurons of the VMH is essential for metabolic responses to fasting and mitochondrial function.

Molecular Metabolism · Feb 8, 2021Peer-reviewedNeuroscience

Dicer-miR-103/107 controls developmental switch of POMC progenitors into NPY neurons and impacts glucose homeostasis

Sophie Croizier, Soyoung Park, Julien Maillard, Sébastien G. Bouret

eLife · Oct 12, 2018Peer-reviewedNeuroscience