Author

André Kleinridders

University of PotsdamORCID 0000-0002-4248-6366Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:metabolomics· 1

Frequent Co-Authors

Attila AndoriD· 1 shared Bernd Timmermann· 1 shared C F SchüßleriD· 1 shared David MeierhoferiD· 1 shared Elias S.J. ArnériD· 1 shared Julia Franke· 1 shared Julia Sophie MeßneriD· 1 shared Kristina WardelmanniD· 1 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperPhysiology

Attila AndorORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Extracellular GPX3 drives insulin receptor expression in adipose tissue independent of its peroxidase activity

Robert Hauffe, Marina Gonzaléz, Simran Chopra, Julia Sophie Meßner, C F Schüßler, Maria Maares, V Kabbouchy, Attila Andor, Elias S.J. Arnér, André Kleinridders

This study identifies GPX3 as a paracrine mediator of adipose tissue insulin sensitivity through a GPX3-SP1-InsR signaling axis, independent of its peroxidase activity.

bioRxiv · Oct 8, 2026 · PreprintPhysiology

Cell autonomous requirement of Neurofibromin (Nf1) for postnatal muscle hypertrophic growth and metabolic homeostasis

Xiaoyan Wei, Julia Franke, Mario Ost, Kristina Wardelmann, Stefan T. Börno, Bernd Timmermann, David Meierhofer, André Kleinridders, Susanne Klaus, Sigmar Stricker

This study shows that Nf1 is required for postnatal muscle growth and metabolic homeostasis, with loss of Nf1 causing muscle atrophy and metabolic changes.

Journal of Cachexia Sarcopenia and Muscle · Oct 19, 2020Peer-reviewedDevelopmental Biology