Author

Jiwon Jung

German Cancer Research CenterORCID 0000-0002-4870-6059Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Jennifer Jung1 paper · The University of Texas at Dallas

Biology Disciplines

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Cancer Biology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:drug response / drug discovery· 2 Assay:bulk RNA-seq· 1 Assay:CRISPR screen· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Barbara M. GrüneriD· 2 shared Madeleine DorschiD· 2 shared Sebastian Urban· 2 shared Ala YarominaiD· 1 shared Alexander Marie-Madeleine Adrianus van der WieliD· 1 shared Alexander Schramm· 1 shared Anna K. Baginska· 1 shared Anne Petzold· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCancer Biology

Barbara M. GrünerORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Cancer BiologybioRxivPreprintOct 8, 2026

Loss of the Y chromosome drives basal identity and metastatic potential via epigenetic reprogramming in pancreatic cancer

Madeleine Dorsch, Felix E.G. Beaudry, Anna K. Baginska…·Barbara M. Grüner

This study identifies LOY as a driver of aggressive PDAC through epigenetic reprogramming, linking it to EMT and metastasis, and highlights its clinical relevance as a biomarker.

DOI 10.64898/2026.10.06.756936

humanbulk RNA-seq

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BiochemistryNature CommunicationsPeer-reviewedMay 13, 2026

Targeted degradation of USP7 in solid cancer cells reveals disparate effects of deubiquitinase inhibition vs. acute protein depletion

Nikolas Klink, Sebastian Urban, Johanna A. Seier…·Malte Gersch

This paper compares the effects of USP7 inhibition and degradation in cancer cells, finding distinct phenotypic and proteomic responses.

1 citation

humanproteomics

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Cancer BiologyMolecular Therapy OncologyPeer-reviewedFeb 24, 2026

Deficiencies in the Fanconi Anemia or the Homologous Recombination Pathway enhance the antitumor effects of the novel hypoxia-activated prodrug CP-506

Lesley Schuitmaker, Alexander Marie-Madeleine Adrianus van der Wiel, Natasja G. Lieuwes…·Philippe Lambin

This study shows that deficiencies in Fanconi Anemia or homologous recombination pathways enhance the antitumor effects of CP-506, a hypoxia-activated prodrug, through synthetic lethality mechanisms.

1 citation

humanexperimental assay

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