Author

Alessandro Sardini

Hammersmith HospitalORCID 0000-0002-4915-3687Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Genetics· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Assay:DNA methylation· 1

Frequent Co-Authors

Amanda G. FisheriD· 2 shared Matthias MerkenschlageriD· 2 shared Zoë WebsteriD· 2 shared Anastasia A. FadeevaiD· 1 shared Andrew DimondiD· 1 shared Anne C. Ferguson‐SmithiD· 1 shared Anthony G. UreniD· 1 shared António Miguel GalvãoiD· 1 shared

Activity by Year

2018-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersGenetics

Amanda G. FisherORCID

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3 papers

Recent·Most cited

GeneticsbioRxivPreprintAug 4, 2025

A step towards animal models with self-sustained fungal bioluminescence

Hannah J. Gleneadie, Nicolás Veland, Eleanor C. Warren…·Amanda G. Fisher

The paper develops a self-sustained fungal bioluminescence system in animal models to monitor molecular events non-invasively.

2 citations

mouseexperimental assay

Developmental BiologybioRxivPreprintAug 7, 2020

Epigenetic change induced by in utero dietary challenge provokes phenotypic variability across multiple generations of mice

Mathew Van de Pette, António Miguel Galvão, Steven J. Millership…·Amanda G. Fisher

The paper shows that maternal high-fat diet induces epigenetic changes in mice that affect two generations, with altered Dlk1 expression and DNA methylation.

2 citations

mouseDNA methylation

Molecular BiologybioRxivPreprintAug 16, 2018

Signalling pathways drive heterogeneity of ground state pluripotency

Kirsten R. McEwen, Sarah Linnett, Harry G. Leitch…·Petra Hájková

This study shows that 2i conditions increase transcriptional heterogeneity in pluripotent stem cells, driven by signaling pathways and negative feedback loss.

2 citations