Author

Samuel Slone

University of CincinnatiORCID 0000-0002-5073-9722Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:cell-cell communication· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1 Organism:rat· 1

Frequent Co-Authors

Jeffrey AubéiD· 2 shared Liang XuiD· 2 shared Lisa C. GreeniD· 2 shared Michael TranteriD· 2 shared Michelle L. NiemaniD· 2 shared Onur KanisicakiD· 2 shared Sarah AnthonyiD· 2 shared Sudeshna RoyiD· 2 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersMolecular Biology

Jeffrey AubéORCID

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2 papers

Recent·Most cited

Molecular BiologybioRxivPreprintJan 17, 2023

HuR inhibition reduces post-ischemic cardiac remodeling by dampening acute inflammatory gene expression and the innate immune response

Samuel Slone, Sarah Anthony, Lisa C. Green…·Michael Tranter

This study shows that inhibiting HuR reduces cardiac remodeling after ischemia-reperfusion injury by dampening inflammatory gene expression and immune response, but not initial infarct size.

5 citations

ratbulk RNA-seq

Molecular BiologyJournal of Molecular and Cellular CardiologyPeer-reviewedJan 1, 2023

HuR-dependent expression of Wisp1 is necessary for TGF????-induced cardiac myofibroblast activity

Lisa C. Green, Samuel Slone, Sarah Anthony…·Michael Tranter

This study shows that HuR-dependent Wisp1 expression is necessary for TGFβ-induced cardiac myofibroblast activity, as HuR inhibition reduces ECM remodeling and contractile functions.

15 citations

mousebulk RNA-seq