Author

Jonathan C. Morris

UNSW SydneyORCID 0000-0002-5109-9069Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Adelaide I.J. YoungiD· 1 shared Amira A.T. Zawia· 1 shared Amy P. Lynch· 1 shared Andrew V. BenestiD· 1 shared Antonia L. CadelliD· 1 shared Benjamin L. ParkeriD· 1 shared Bruce Braithwaite· 1 shared Chiara PantarelliiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCancer Biology

Adelaide I.J. YoungORCID

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2 papers

Recent·Most cited

PhysiologyAmerican Journal of Physiology-Heart and Circulatory PhysiologyPeer-reviewedJun 30, 2025

The splicing factor kinase SRPK1 is a therapeutic target for Peripheral Vascular Disease

Sohni Ria Bhalla, Mussarat Wahid, Jason O. Amartey…·David O. Bates

This paper shows that inhibiting SRPK1 in monocytes promotes angiogenesis by altering VEGF splicing from anti-angiogenic VEGF-A165b to pro-angiogenic VEGF-A165a in both human and mouse models of peripheral arterial disease.

5 citations

mousehuman

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Cancer BiologybioRxivPreprintApr 16, 2025

A selective inhibitor of oncogenic JNK signalling perturbs metastatic outgrowth of triple-negative breast cancer through metabolic blockade

Sharissa L. Latham, Yolande E. I. O’Donnell, Oscar Girgis-Cook…·David R. Croucher

The paper identifies a selective JNK inhibitor that blocks metastasis in triple-negative breast cancer by targeting metabolic pathways.

DOI 10.1101/2025.04.16.649243

humanproteomics

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