Daniel B. Polley

The paper identifies neural and autonomic predictors of future listening errors in a sustained auditory task.

The paper distinguishes between spontaneous and stimulus-driven acetylcholine dynamics in sensory and prefrontal cortex using genetically encoded sensors and multivariate modeling in awake mice.

This study shows that enhancing inhibition in the auditory cortex can reverse maladaptive plasticity in the amygdala caused by noise-induced hearing loss.

The paper investigates whether neural and behavioral markers of distraction suppression correlate with tinnitus severity, finding no such association.

The paper identifies layer 1 inhibitory neurons and layer 6 excitatory neurons as hubs mediating cholinergic modulation in the mouse auditory cortex through distinct receptor mechanisms.

This paper measures neural synchronization to different sound timescales in human listeners to determine how distraction affects auditory processing.

This study shows that age-related changes in central arousal states, measured via pre-stimulus pupil size, explain moment-to-moment fluctuations in speech intelligibility in older adults.

This study shows that modulating parvalbumin-expressing neurons in the auditory cortex can adjust loudness perception and reverse auditory hypersensitivity in mice.

This study shows that basal forebrain cholinergic activation modulates amygdala neuron activity based on reward contingency, affecting conditioned behavior but not unconditioned responses.

The paper identifies objective signatures of tinnitus and auditory hypersensitivity disorders through pupil dilation and facial movement analysis in response to sound valence.

The paper identifies facial motion energy (FME) as a sensitive measure of hearing abilities in mice, showing its reliability and ability to decode speech in noise.

This study investigates how changes in parvalbumin-expressing inhibitory interneurons in the auditory cortex affect loudness perception and hyperacusis in mice.

The paper shows that during threat learning, BLA cholinergic inputs and corticofugal projections are reorganized to support sensory processing changes in mice.

This study shows that acoustic trauma leads to non-homeostatic gain changes in auditory cortex neurons, contributing to hypersensitivity to sound.

The paper identifies a functional topography in basal forebrain cholinergic neurons that differentiates their responses to sensory cues and reinforcement outcomes in mice.

The paper identifies that motor preparatory inputs activate auditory corticothalamic neurons before orofacial movements in mice.

The paper identifies neural signatures of poor multi-talker speech perception in adults with normal hearing, linking it to phase locking to frequency modulation cues and cognitive listening effort.

The paper uses imaging techniques to map sound frequency organization in mouse auditory cortex fields, identifying new areas and clarifying tonotopic organization.

The paper optimizes optogenetic stimulation protocols in auditory corticofugal neurons using closed-loop spike feedback to enhance or suppress midbrain firing rates.

This study shows that cholinergic basal forebrain neurons enable auditory cortex reorganization for learning by bridging temporal gaps between sensory stimuli and delayed reinforcement.

The paper investigates functional connectivity and sensory processing differences between layer 5 and 6 corticothalamic neurons in the mouse auditory cortex using optogenetic recordings.

The paper shows that auditory cortex projection neurons adjust their gain after cochlear nerve damage, leading to increased excitability and potential network dysfunction in tinnitus.