Marco B. Rust

This study identifies CAP1 as a repressor of MRTF-SRF signaling in mouse neurons through actin-dependent mechanisms and validates its role in gene regulation in the cerebral cortex.

This study shows that cofilin1 oxidation contributes to mitochondrial dysfunction and neuronal death by linking oxidative stress to ferroptosis and excitotoxicity.

The paper identifies CAP1 as a novel actin regulator in growth cones that is critical for neuron connectivity and function.

This study identifies CAP2 as a regulator of Cofilin in synaptic plasticity, showing its role in LTP and its disruption in Alzheimer’s disease.