Author

Trent E. Tipple

University of Oklahoma Harold Hamm Health Campus;ORCID 0000-0002-5156-4282Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Organism:mouse· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Lynette Kay RogersiD· 3 shared Abhrajit GangulyiD· 2 shared Aristides Rivera Negron· 2 shared Cynthia M. Carter· 2 shared Matthew S. WaltersiD· 2 shared Y. S. PrakashiD· 2 shared Alvaro G. MoreiraiD· 1 shared Arlan G RichardsoniD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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Curved chords = mutual co-authorship

3 shared papersDevelopmental Biology

Lynette Kay RogersORCID

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Author profile →

3 papers

Recent·Most cited

Endothelial Cell Specific Thioredoxin 1-Dependent Signaling in Oxygen-1 Induced Retinopathy

Henry Song, Wenjing Wu, Daniyal Khan, Hua Zhong, Mathew Meaders, Paul T Pierce, Peter F Vitiello, Lynette Rogers, Trent E Tipple, Faizah N Bhatti

This study shows that endothelial cell-specific TXN1 protects against oxygen-induced retinopathy by regulating proliferation and redox signaling.

bioRxiv · Nov 6, 2026 · PreprintCell Biology

3-Mercaptopyruvate Sulfurtransferase (MPST) Regulates Mitochondrial Metabolism and Epithelial Differentiation in Neonatal Patient-derived Airway Cells

Abhrajit Ganguly, Cynthia M. Carter, Aristides Rivera Negron, Paul T. Pierce, Lynette Kay Rogers, Matthew S. Walters, Y. S. Prakash, Trent E. Tipple, Arlan G Richardson

This study shows that MPST is essential for maintaining mitochondrial metabolic integrity in neonatal airway epithelial cells, and its loss under hyperoxia disrupts epithelial differentiation and contributes to airway injury.

American Journal of Physiology-Lung Cellular and Molecular Physiology · Mar 28, 2026Peer-reviewedDevelopmental Biology

Quantitative Proteomics Links Mitochondrial Dysfunction to Metabolic Changes and Epithelial Differentiation Defects in Hyperoxia-Exposed Neonatal Airway Cells

Abhrajit Ganguly, Cynthia M. Carter, Aristides Rivera Negron, Hua Zhong, Alvaro G. Moreira, Matthew S. Walters, Lynette Kay Rogers, Y. S. Prakash, Trent E. Tipple, Arlan Richardson

This study shows that hyperoxia impairs mitochondrial function and epithelial differentiation in neonatal airway cells through proteomic analysis and metabolic reprogramming.

American Journal of Physiology-Lung Cellular and Molecular Physiology · Dec 5, 2025Peer-reviewedDevelopmental Biology