Author

Peder Berg

Aarhus UniversityORCID 0000-0002-5216-2891Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Albert C.M. OngiD· 1 shared Bolette HartmanniD· 1 shared Helga VitzthumiD· 1 shared Ida Marie ModvigiD· 1 shared Jens Juul HolstiD· 1 shared Jens LeipzigeriD· 1 shared Jesper Frank AnderseniD· 1 shared Laura Woidemann Trans· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperCell Biology

Albert C.M. OngORCID

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2 papers

Recent·Most cited

Impaired renal base excretion in secretin receptor knock-out mice during prolonged base-loading

Tobias Jensen, Jesper Frank Andersen, Laura Woidemann Trans, Ida Marie Modvig, Helga Vitzthum, Jens Juul Holst, Bolette Hartmann, Samuel L. Svendsen, Mads Vaarby Sørensen, Jens Leipziger, Peder Berg

This study shows that secretin receptor knockout mice have impaired renal base excretion and increased base retention during prolonged base-loading.

Acta Physiologica · Jul 31, 2026Peer-reviewedPhysiology

Polycystin-1 acts as an atypical adhesion GPCR that responds to novel Wnt signaling and mechanical stimuli

Nikolay Gresko, Valeria Padovano, Peder Berg, Albert C.M. Ong, Stefan Somlo, Nicole Scholz, Tobias Langenhan, Michael J. Caplan

This paper identifies Polycystin-1 as an atypical adhesion GPCR that activates a signaling pathway through Wnt ligands and mechanical stimuli, leading to GSK3 inhibition and ciliary removal.

bioRxiv · May 21, 2025 · PreprintCell Biology