Author

Nicola C. Partridge

Rutgers, The State University of New JerseyORCID 0000-0002-5406-4814Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Carole Le HénaffiD· 2 shared Zhiming He· 2 shared Aditya Mohapatra· 1 shared Despina SitaraiD· 1 shared Henry M. KronenbergiD· 1 shared Jennifer J. WestendorfiD· 1 shared Jobin Joseph· 1 shared Johanna WarshawiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersPhysiology

Carole Le HénaffORCID

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2 papers

Recent·Most cited

PhysiologybioRxivPreprintSep 27, 2026

The CREB-regulated co-activators 2/3, have a role, in vivo, in osteoblastic gene expression.

Carole Le Hénaff, Jobin Joseph, Whitney Petrosky…·Nicola C. Partridge

This study investigates the in vivo role of CRTC2/3 in osteoblasts by analyzing bone phenotype and gene expression changes in conditional knockout mice.

DOI 10.64898/2026.09.21.753306

mouseexperimental assay

𝕏 PostBluesky

PathologybioRxivPreprintMar 13, 2026

Inducible activation of PKA in osteoblasts causes a profound high bone turnover phenotype similar to human diseases

Carole Le Hénaff, Zhiming He, Joshua H. Johnson…·Nicola C. Partridge

This study shows that inducible PKA activation in osteoblasts leads to high bone turnover and disease-like phenotypes in mice.

DOI 10.64898/2026.03.11.709826

mouseexperimental assay

𝕏 PostBluesky