Author

Zhiming He

New York UniversityPrimary ORCID profile (0000-0002-6011-3838 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Zhiming HeiD1 paper · Hunan University of Humanities, Science and Technology

Biology Disciplines

Click to filter author papers

Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Carole Le HénaffiD· 2 shared Nicola C. PartridgeiD· 2 shared Aditya Mohapatra· 1 shared Despina SitaraiD· 1 shared Henry M. KronenbergiD· 1 shared Jennifer J. WestendorfiD· 1 shared Jobin Joseph· 1 shared Johanna WarshawiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersPhysiology

Carole Le HénaffORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

The CREB-regulated co-activators 2/3, have a role, in vivo, in osteoblastic gene expression.

Carole Le Hénaff, Jobin Joseph, Whitney Petrosky, Zhiming He, Aditya Mohapatra, Sohan Sirimarapu, Jennifer J. Westendorf, Marc A Montminy, Nicola C. Partridge

This study investigates the in vivo role of CRTC2/3 in osteoblasts by analyzing bone phenotype and gene expression changes in conditional knockout mice.

bioRxiv · Sep 27, 2026 · PreprintPhysiology

Inducible activation of PKA in osteoblasts causes a profound high bone turnover phenotype similar to human diseases

Carole Le Hénaff, Zhiming He, Joshua H. Johnson, Johanna Warshaw, Rocco Latorre, Nigel W. Bunnett, Despina Sitara, Lawrence Steven Kirschner, Henry M. Kronenberg, Nicola C. Partridge

This study shows that inducible PKA activation in osteoblasts leads to high bone turnover and disease-like phenotypes in mice.

bioRxiv · Mar 13, 2026 · PreprintPathology