Author

Rúben G. R. Pinheiro

Gulbenkian Institute for Molecular MedicineORCID 0000-0002-5475-4719Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

Adrian HaydayiD· 1 shared Ana Redondo‐UrzainquiiD· 1 shared Andrew S. MacDonaldiD· 1 shared Ángela Zarco-CuadrilleroiD· 1 shared Bruno Silva‐SantosiD· 1 shared David SanchoiD· 1 shared Elena Hernández-GarciaiD· 1 shared James E ParkinsoniD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperImmunology

Adrian HaydayORCID

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1 paper

Recent·Most cited

BATF3 controls the homeostatic maintenance and function of innate-like γδ T cells

Pedro Henrique Papotto, Elena Hernández-Garcia, Ángela Zarco-Cuadrillero, Ana Redondo‐Urzainqui, James E Parkinson, Rúben G. R. Pinheiro, Andrew S. MacDonald, David Sancho, Bruno Silva‐Santos, Judith E. Allen, Adrian Hayday, Salvador Iborra, Miguel Muñoz‐Ruiz

This study identifies BATF3 as a critical regulator of innate-like γδ T cell homeostasis, function, and tissue distribution, revealing its role in survival, IL-17 production, and immune responses.

bioRxiv · Dec 18, 2025 · PreprintImmunology