Author

Oto Inoue

Cincinnati Children's Hospital Medical CenterORCID 0000-0002-5559-8981Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:variant calling· 1

Frequent Co-Authors

Hee-Woong LimiD· 2 shared Joan Sanchez-GurmachesiD· 2 shared Archana Singh· 1 shared Christian Louis Bonatto PaeseiD· 1 shared Christopher Ahn· 1 shared Manasi Suchit HalurkariD· 1 shared Marine A Secchi· 1 shared Meghana GinuguiD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersDevelopmental Biology

Hee-Woong LimORCID

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2 papers

Recent·Most cited

Unbiased lineage tracing across the adipose organ decouples developmental origin from anatomical and transcriptomic identities

Oto Inoue, Archana Singh, Meghana Ginugu, Christopher Ahn, Marine A Secchi, Sarah J Bowling, Hee-Woong Lim, Joan Sanchez-Gurmaches

This study uses CRISPR-mediated lineage tracing to show that adipose depot identity is shaped by anatomical niche rather than embryonic lineage.

bioRxiv · Nov 9, 2026 · PreprintDevelopmental Biology

The widely used Ucp1-CreEvdr transgene elicits complex developmental and metabolic phenotypes.

Manasi Suchit Halurkar, Oto Inoue, Rajib Mukherjee, Christian Louis Bonatto Paese, Molly Duszynski, Samantha A. Brugmann, Hee‐Woong Lim, Joan Sánchez-Gurmaches

The paper analyzes the Ucp1-CreEvdr transgenic mouse line and finds it causes complex developmental and metabolic issues independent of its intended genetic manipulation.

bioRxiv · Oct 20, 2023 · PreprintMolecular Biology