Author

Pietro Luigi Poliani

Ospedale Santa ChiaraORCID 0000-0002-5662-8978Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1 Approach:microscopy & imaging analysis· 1

Frequent Co-Authors

Alessandro QuattroneiD· 1 shared Alessia InglesiiD· 1 shared Alessia SoldanoiD· 1 shared Alfredo BerrutiiD· 1 shared Andrea AbateiD· 1 shared Chiara Maria ArgentoiD· 1 shared Claudia Bonera· 1 shared Deborah CosentiniiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

Alessandro QuattroneORCID

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2 papers

Recent·Most cited

Updating ACC preclinical models: characterization of two new patient-derived cell lines

Andrea Abate, Mariangela Tamburello, Claudia Bonera, Guido Alberto Massimo Tiberio, Sara Baldelli, Mattia Carini, Alessia Inglesi, Maria Scatolini, Deborah Cosentini, Marta Laganà, Marta Leporati, Pietro Luigi Poliani, Domenico Cosseddu, Duilio Brugnoni, Alfredo Berruti, Sandra Sigala

This paper describes the development and characterization of two new patient-derived ACC cell lines, SMAC-2 and SMAC-3, highlighting their molecular and functional features for preclinical research.

Endocrine Related Cancer · May 1, 2026Peer-reviewedPharmacology & Toxicology

Unveiling Oligodendrocyte-Lineage Differentiation in the Glioblastoma Infiltrative Zone Through Spatial Transcriptomics

Chiara Maria Argento, Enrico Sebastiani, Emanuele Filiberto Rosatti, Denise Sighel, Sara Longhi, Alessia Soldano, Toma Tebaldi, Alessandro Quattrone, Francesco Corsini, Luciano Annicchiarico, Mattia Barbareschi, Lorenza Pecciarini, Pietro Luigi Poliani

This study uses spatial transcriptomics to show that infiltrative glioblastoma cells differentiate toward an oligodendrocyte-like state, which may contribute to tumor invasion and recurrence.

bioRxiv · Oct 20, 2025 · PreprintCancer Biology