Author

Miguel Angel Abellanas

Weizmann Institute of ScienceORCID 0000-0002-5680-1582Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Hannah Partney· 3 shared Yuliya Androsova· 3 shared Angham Ibraheem· 2 shared Bar Nathansohn· 2 shared Giulia CastellaniiD· 2 shared Javier María Peralta RamosiD· 2 shared Michal SchwartziD· 2 shared Sama Murad· 2 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersNeuroscience

Hannah Partney

Mutual co-authors in cluster

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3 papers

Recent·Most cited

NeuroscienceNature CommunicationsPeer-reviewedJun 19, 2026

Disruption of the Brain-Spleen Axis Impairs Monocyte-Microglia Communication and Accelerates Disease Progression in a Model of Amyloidosis

Tommaso Croese, Miguel Angel Abellanas, Hodaya Polonsky…·Michal Schwartz

This study shows that disrupting the brain-spleen axis accelerates disease progression in amyloidosis by impairing monocyte-microglia communication.

1 citation

mouseexperimental assay

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NeurosciencebioRxivPreprintDec 24, 2025

Systemic administration of PD-L1 blocking antibodies leads to removal of senescent microglia

Yuliya Androsova, Alexander Kertser, Hannah Partney…·Michal Schwartz

The paper shows that systemic PD-L1 blockade removes senescent microglia in mice, reducing neuroinflammation and suggesting a therapeutic role in Alzheimer's disease.

1 citation

mouseexperimental assay

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NeurosciencebioRxivPreprintOct 6, 2025

Alzheimer's disease causes bone marrow myelopoiesis dysfunction

Miguel Angel Abellanas, Leyre Basurco, Maitreyee Purnapatre…·Michal Schwartz

This study identifies impaired bone marrow myelopoiesis in Alzheimer's disease, driven by type I interferon signaling, and shows that targeting this pathway improves myeloid function and cognitive outcomes.

3 citations

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