Author

Giulia Castellani

Weizmann Institute of ScienceORCID 0000-0002-0611-5250Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Hannah Partney· 2 shared Javier María Peralta RamosiD· 2 shared Miguel Angel AbellanasiD· 2 shared Sama Murad· 2 shared Sarah Phoebeluc ColaiutaiD· 2 shared Tomer‐Meir SalameiD· 2 shared Yuliya Androsova· 2 shared Aleksandra DeczkowskaiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

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2 shared papersNeuroscience

Hannah Partney

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Disruption of the Brain-Spleen Axis Impairs Monocyte-Microglia Communication and Accelerates Disease Progression in a Model of Amyloidosis

Tommaso Croese, Miguel Angel Abellanas, Hodaya Polonsky, Michal Arad, Javier María Peralta Ramos, Yuliya Androsova, Serena Riccitelli, Sedi Medina, Francesca Palmas, Romano Strobelt, Giulia Castellani, Denise Kviatcovsky, Sarah Phoebeluc Colaiuta, Miriam Adam, Sama Murad, , Michal Schwartz

This study shows that disrupting the brain-spleen axis accelerates disease progression in amyloidosis by impairing monocyte-microglia communication.

Nature Communications · Jun 19, 2026Peer-reviewedNeuroscience

Alzheimer's disease causes bone marrow myelopoiesis dysfunction

Miguel Angel Abellanas, Leyre Basurco, Maitreyee Purnapatre, Chiara Burgaletto, Giulia Castellani, Sarah Phoebeluc Colaiuta, Javier María Peralta Ramos, Angham Ibraheem, Sama Murad, Paola Antonello, Mariangeles Kovacs, Yuliya Androsova, Bar Nathansohn, Hannah Partney, Liora Cahalon, , Michal Schwartz

This study identifies impaired bone marrow myelopoiesis in Alzheimer's disease, driven by type I interferon signaling, and shows that targeting this pathway improves myeloid function and cognitive outcomes.

bioRxiv · Oct 6, 2025 · PreprintNeuroscience