Author

Yuliya Androsova

Weizmann Institute of Science

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Hannah Partney· 3 shared Miguel Angel AbellanasiD· 3 shared Angham Ibraheem· 2 shared Bar Nathansohn· 2 shared Giulia CastellaniiD· 2 shared Javier María Peralta RamosiD· 2 shared Michal SchwartziD· 2 shared Sama Murad· 2 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersNeuroscience

Hannah Partney

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Disruption of the Brain-Spleen Axis Impairs Monocyte-Microglia Communication and Accelerates Disease Progression in a Model of Amyloidosis

Tommaso Croese, Miguel Angel Abellanas, Hodaya Polonsky, Michal Arad, Javier María Peralta Ramos, Yuliya Androsova, Serena Riccitelli, Sedi Medina, Francesca Palmas, Romano Strobelt, Giulia Castellani, Denise Kviatcovsky, Sarah Phoebeluc Colaiuta, Miriam Adam, Sama Murad, , Michal Schwartz

This study shows that disrupting the brain-spleen axis accelerates disease progression in amyloidosis by impairing monocyte-microglia communication.

Nature Communications · Jun 19, 2026Peer-reviewedNeuroscience

Systemic administration of PD-L1 blocking antibodies leads to removal of senescent microglia

Yuliya Androsova, Alexander Kertser, Hannah Partney, Bar Nathansohn, Angham Ibraheem, Miguel Angel Abellanas, Tommaso Croese, Tomer Meir Salame, Hagay Akiva, Valery Krizhanovsky, Michal Schwartz

The paper shows that systemic PD-L1 blockade removes senescent microglia in mice, reducing neuroinflammation and suggesting a therapeutic role in Alzheimer's disease.

bioRxiv · Dec 24, 2025 · PreprintNeuroscience

Alzheimer's disease causes bone marrow myelopoiesis dysfunction

Miguel Angel Abellanas, Leyre Basurco, Maitreyee Purnapatre, Chiara Burgaletto, Giulia Castellani, Sarah Phoebeluc Colaiuta, Javier María Peralta Ramos, Angham Ibraheem, Sama Murad, Paola Antonello, Mariangeles Kovacs, Yuliya Androsova, Bar Nathansohn, Hannah Partney, Liora Cahalon, , Michal Schwartz

This study identifies impaired bone marrow myelopoiesis in Alzheimer's disease, driven by type I interferon signaling, and shows that targeting this pathway improves myeloid function and cognitive outcomes.

bioRxiv · Oct 6, 2025 · PreprintNeuroscience