Author

Alexandra Michurina

German Center for Neurodegenerative DiseasesORCID 0000-0002-5704-2282Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Assay:ChIP-seq· 3 Research Focus:differential expression· 3 Organism:mouse· 3 Approach:statistical model· 3 Assay:bulk RNA-seq· 2 Research Focus:gene regulatory network inference· 2 Research Focus:cell type annotation· 1 Approach:experimental assay· 1

Frequent Co-Authors

André FischeriD· 3 shared Cemil KerimogluiD· 3 shared Lalit KauraniiD· 3 shared M. Sadman SakibiD· 3 shared Andrea KranziD· 2 shared Dennis Manfred Krüger· 2 shared Elisabeth Maria ZeisbergiD· 2 shared Julia Cha· 2 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersNeuroscience

André FischerORCID

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3 papers

Recent·Most cited

Developmental BiologyScience AdvancesPeer-reviewedSep 15, 2021

H3 acetylation selectively promotes basal progenitor proliferation and neocortex expansion by activating TRNP1 expression

Cemil Kerimoglu, Linh Pham, Anton B. Tonchev…·Tran Tuoc

The paper shows H3K9ac increases basal progenitor proliferation and neocortex size by upregulating TRNP1 in evolution.

29 citations

𝕏 PostBluesky

NeurosciencebioRxivPreprintAug 2, 2021

Postnatal SETD1B is essential for learning and the regulation of neuronal plasticity genes

Alexandra Michurina, M. Sadman Sakib, Cemil Kerimoglu…·André Fischer

This study shows that SETD1B is crucial for learning and regulates neuronal plasticity genes in the postnatal brain through H3K4me3 modification.

DOI 10.1101/2021.08.02.454636

𝕏 PostBluesky

NeurosciencebioRxivPreprintAug 7, 2020

SETD1B controls cognitive function via cell type specific regulation of neuronal identity genes

Alexandra Michurina, M. Sadman Sakib, Cemil Kerimoglu…·André Fischer

The paper shows that Setd1b regulates neuronal identity genes in the adult brain, affecting cognitive function and memory, with other KMTs compensating for its absence.

DOI 10.1101/2020.08.07.240853

𝕏 PostBluesky