Shubhra Majumder

This study shows that cytoplasmic recapping of MALAT1 produces the micropeptide MARS, which is regulated by RNA G-quadruplex structures and involved in oxidative stress response.

This study shows that low-dose rotenone exposure causes primary cilia elongation and dysfunction in quiescent cells, independent of mitochondrial toxicity.

The paper identifies a pathway where tubulin hyperacetylation and ROS-PARP1 signaling drive HMGB1 nuclear exit, leading to G2/M arrest induced by rotenone.

The paper investigates the role of cytoplasmic capping enzyme (cCE) in maintaining cap homeostasis of target mRNAs during stress and recovery.