Cell BiologyJournal of Biological ChemistryPeer-reviewed
Tubulin hyperacetylation drives HMGB1 nuclear exit via the ROS-PARP1 axis leading to rotenone-induced G2/M Arrest
Sourav Dutta, Semanti Chakraborty, Ayushi Ghosh…·Piyali Mukherjee
The paper identifies a pathway where tubulin hyperacetylation and ROS-PARP1 signaling drive HMGB1 nuclear exit, leading to G2/M arrest induced by rotenone.
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