Author

Piyali Mukherjee

Presidency UniversityORCID 0000-0002-0140-7414Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Organism:mouse· 1 Research Focus:transcription & RNA processing· 1

Frequent Co-Authors

Abhik SahaiD· 2 shared Joyanta BiswasiD· 2 shared Semanti ChakrabortyiD· 2 shared Sourav DuttaiD· 2 shared Akasmika PriyadarsaniiD· 1 shared Anirban BasuiD· 1 shared Ayushi GhoshiD· 1 shared Debanjan MukhopadhyayiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersImmunology

Abhik SahaORCID

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3 papers

Recent·Most cited

Gasdermin E couples mitochondrial stress to STING-driven neuronal pyroptosis during Chandipura virus encephalitis

Nripa Kumari, Sourav Dutta, Akasmika Priyadarsani, Joyanta Biswas, Shubhangi Roy, Suman Samanta, Semanti Chakraborty, Jyotirmoyee Roy, Anirban Basu, Debanjan Mukhopadhyay, Abhik Saha, Piyali Mukherjee

This study identifies a mitochondria-GSDME-STING axis that links early immune evasion to neuroinflammation during Chandipura virus infection, revealing a mechanism of inflammatory neuronal death.

bioRxiv · Sep 27, 2026 · PreprintImmunology

Tubulin hyperacetylation drives HMGB1 nuclear exit via the ROS-PARP1 axis leading to rotenone-induced G2/M Arrest

Sourav Dutta, Semanti Chakraborty, Ayushi Ghosh, Priyadarshini Halder, Shubhra Majumder, Ratnadip Paul, Somsubhra Nath, Piyali Mukherjee

The paper identifies a pathway where tubulin hyperacetylation and ROS-PARP1 signaling drive HMGB1 nuclear exit, leading to G2/M arrest induced by rotenone.

Journal of Biological Chemistry · Sep 8, 2025Peer-reviewedCell Biology

E2F1 Suppresses EBV Lytic Reactivation through Cellular and Viral Transcriptional Networks

Joyanta Biswas, SK Asif Ali, Samaresh Malik, Subhadeep Nag, Piyali Mukherjee, Abhik Saha

This study identifies E2F1 as a suppressor of EBV lytic reactivation by regulating transcriptional networks involving BZLF1 and c-Myc.

bioRxiv · Jan 23, 2025 · PreprintCancer Biology