Author

Timothy W. Church

University College LondonORCID 0000-0002-5958-6304Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:protein structure· 1

Frequent Co-Authors

Matthew G. GoldiD· 2 shared Ashton J. CurtisiD· 1 shared Dorota StudniarczykiD· 1 shared Ian J. White· 1 shared Jian Zhu· 1 shared Mark FarrantiD· 1 shared Ryan S. DowselliD· 1 shared Yuelin LuoiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 16 mutual co-author links

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2 shared papersMolecular Biology

Matthew G. GoldORCID

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2 papers

Recent·Most cited

Molecular BiologybioRxivPreprintAug 15, 2026

PKA can adopt structural or enzymatic roles depending on its anchoring site

Timothy W. Church, Yuelin Luo, Ryan S. Dowsell…·Matthew G. Gold

This study shows that PKA can act as either an enzymatic or structural component depending on its anchoring site, using a SpyCatcher-SpyTag strategy to compare AKAP functions.

DOI 10.64898/2026.08.12.743961

protein structureexperimental assay

𝕏 PostBluesky

NeuroscienceJournal of NeurosciencePeer-reviewedOct 23, 2025

Widely used CaMKII regulatory segment mutations cause tight actinin binding and dendritic spine enlargement in unstimulated neurons

Ashton J. Curtis, Jian Zhu, Dorota Studniarczyk…·Matthew G. Gold

This study shows that CaMKII T305A/T306A mutations cause tight actinin binding and dendritic spine enlargement in unstimulated neurons, suggesting a structural role for CaMKII in LTP.

DOI 10.1523/jneurosci.0795-25.2025

experimental assay

𝕏 PostBluesky