Author

Mengying Liu

Xiamen UniversityORCID 0000-0002-5987-1287Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:metabolomics· 1

Frequent Co-Authors

B H HeiD· 1 shared Caihe WeniD· 1 shared Dan ZhangiD· 1 shared Dandan BaiiD· 1 shared Dandan Chen· 1 shared Haibin WangiD· 1 shared Haiyan Wen· 1 shared Jinhua LuiD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperDevelopmental Biology

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2 papers

Recent·Most cited

Metabolic adaptation to maternal hyperglycemia via ACLY-dependent acetyl-CoA production drives epigenetic remodeling and dysregulated placental development

Mengying Liu, Keer Jin, Shihong Qi, Dandan Chen, Yichen Han, Weina Xu, Caihe Wen, Haiyan Wen, Yuanwei Liu, B H He, Xiaona Lin

The paper identifies ACLY-dependent acetyl-CoA production as a mechanism linking maternal hyperglycemia to epigenetic remodeling and placental underdevelopment in gestational diabetes.

bioRxiv · Mar 20, 2026 · PreprintDevelopmental Biology

Maternal Ezh1/2 deficiency in oocyte delays H3K27me2/3 restoration and impairs epiblast development responsible for embryonic sub-lethality in mouse

Yinan Zhao, Dandan Bai, You Wu, Dan Zhang, Mengying Liu, Yingpu Tian, Jinhua Lu, Haibin Wang, Shaorong Gao, Zhongxian Lu

This study shows that maternal Ezh1/2 deficiency disrupts H3K27 methylation in oocytes, leading to delayed restoration and impaired epiblast development, causing embryonic sub-lethality in mice.

Development · Jul 7, 2022Peer-reviewedDevelopmental Biology