Author

Alison M. Schram

Memorial Sloan Kettering Cancer CenterORCID 0000-0002-6070-2413Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:benchmark study· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:human· 1 Research Focus:perturbation response prediction· 1

Frequent Co-Authors

Britta WeigeltiD· 1 shared Elisa de StanchinaiD· 1 shared Harika GundlapalliiD· 1 shared Heeseon AniD· 1 shared Hilla SolomoniD· 1 shared Jacqueline A.M. SmithiD· 1 shared Julia Meredith· 1 shared Justin MeyerowitziD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

Britta WeigeltORCID

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1 paper

Recent·Most cited

Cancer BiologybioRxivPreprintFeb 14, 2026

Coexistent PTEN and PIK3CA alterations hyperactivate mTORC1 signaling in endometrial cancers and cause their selective sensitivity to mTORC1 inhibition

Hilla Solomon, Radha Mukherjee, Yu Chi Yang…·Neal X. Rosen

This study shows that coexisting PTEN and PIK3CA mutations in endometrial cancers hyperactivate mTORC1, making them sensitive to mTORC1 inhibitors but not PI3K or AKT inhibitors.

DOI 10.64898/2026.02.12.705558

humanbulk RNA-seq

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