Author

Hilla Solomon

Memorial Sloan Kettering Cancer CenterORCID 0000-0003-1712-3202Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:benchmark study· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Elisa de StanchinaiD· 2 shared Neal X. RoseniD· 2 shared Radha MukherjeeiD· 2 shared Alison M. SchramiD· 1 shared Britta WeigeltiD· 1 shared Claire N. ThantiD· 1 shared Harika GundlapalliiD· 1 shared Heeseon AniD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersCancer Biology

Elisa de StanchinaORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintFeb 14, 2026

Coexistent PTEN and PIK3CA alterations hyperactivate mTORC1 signaling in endometrial cancers and cause their selective sensitivity to mTORC1 inhibition

Hilla Solomon, Radha Mukherjee, Yu Chi Yang…·Neal X. Rosen

This study shows that coexisting PTEN and PIK3CA mutations in endometrial cancers hyperactivate mTORC1, making them sensitive to mTORC1 inhibitors but not PI3K or AKT inhibitors.

DOI 10.64898/2026.02.12.705558

humanbulk RNA-seq

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Cell BiologybioRxivPreprintMar 25, 2025

Diet induced insulin resistance is due to induction of PTEN expression

Radha Mukherjee, Priya Pancholi, Malvika Sharma…·Neal X. Rosen

The paper shows that diet-induced insulin resistance results from increased PTEN expression, which inhibits PI3K signaling, and that PTEN and mTORC1 inhibitors can reverse this effect.

2 citations

mousestatistical model

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