Author

Wei Huang

University of North Carolina at Chapel HillORCID 0000-0002-6081-7878Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Wei Huang9 papers · University of Electronic Science and Technology of China

Biology Disciplines

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Molecular Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

Alicia Newton· 1 shared Andrew L. Chin· 1 shared Ayşegül ÖzeniD· 1 shared Brett R. ChrestiD· 1 shared Brian C. JenseniD· 1 shared Celia A. SchifferiD· 1 shared Christos J. PetropoulosiD· 1 shared Cihan AydıniD· 1 shared

Activity by Year

2018-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperMolecular Biology

Alicia Newton

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2 papers

Recent·Most cited

PhysiologyScience AdvancesPeer-reviewedJul 10, 2026

The MEK inhibitor trametinib incurs mitochondrial injury and induces innate immune responses in the mouse heart

Kelsey H. Fisher‐Wellman, Richard D. Lutze, Logan G. Kirkland…·Brian C. Jensen

This study shows trametinib causes mitochondrial injury and innate immune activation in mouse hearts, leading to cardiomyopathy and heart failure.

DOI 10.1126/sciadv.aeb2695

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Molecular BiologybioRxivPreprintDec 16, 2018

Resistance from Afar: Distal Mutation V36M Allosterically Modulates the Active Site to Accentuate Drug Resistance in HCV NS3/4A Protease

Ayşegül Özen, Kuan-Hung Lin, Keith P. Romano…·Celia A. Schiffer

This study investigates how the distal mutation V36M confers drug resistance in HCV NS3/4A protease through allosteric modulation of the active site, in the context of R155K.

1 citation

experimental assay

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