Author

Feras E. Machour

Technion – Israel Institute of TechnologyORCID 0000-0002-6446-0474Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Feras E. Machour1 paper · The Francis Crick Institute

Biology Disciplines

Click to filter author papers

Molecular Biology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Organism:human· 3 Research Focus:differential expression· 2 Assay:DNA methylation· 2 Research Focus:gene regulatory network inference· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Assay:ChIP-seq· 1

Frequent Co-Authors

Enas R. Abu‐Zhayia· 3 shared Nabieh AyoubiD· 3 shared Alma Sophia Barisaac· 2 shared Laila A. BisharaiD· 2 shared Bella M. Ben‐Oz· 1 shared Christeen Elmor· 1 shared Itamar SimoniD· 1 shared Joyce KamariD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 25 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersMolecular Biology

Enas R. Abu‐Zhayia

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

LSm4 biomolecular condensates drive XRN2-mediated RNA decay at DNA double-strand breaks to facilitate repair

Malak M. Darawshe, Laila A. Bishara, Enas R. Abu‐Zhayia, Alma Sophia Barisaac, Feras E. Machour, Christeen Elmor, Nabieh Ayoub

This study identifies LSm4 biomolecular condensates as key regulators of RNA decay at DNA double-strand breaks, preventing R-loop accumulation and enabling efficient repair.

bioRxiv · Aug 20, 2026 · PreprintMolecular Biology

Harnessing DNA replication stress to target RBM10 deficiency in lung adenocarcinoma

Feras E. Machour, Enas R. Abu‐Zhayia, Joyce Kamar, Alma Sophia Barisaac, Itamar Simon, Nabieh Ayoub

This study identifies RBM10's role in DNA replication and shows that targeting WEE1 can sensitize RBM10-deficient lung adenocarcinoma cells.

Nature Communications · Jul 30, 2024Peer-reviewedMolecular Biology

Systematic identification of a CDYL1-dependent decrease in lysine crotonylation at DNA double-strand break sites functionally uncouples transcriptional silencing and repair

Enas R. Abu‐Zhayia, Feras E. Machour, Laila A. Bishara, Bella M. Ben‐Oz, Nabieh Ayoub

The paper identifies CDYL1-dependent reduction in lysine crotonylation at DSBs, showing transcriptional silencing and repair are functionally uncoupled.

bioRxiv · May 31, 2021 · PreprintMolecular Biology