Author

Guvem Gumus Akay

Ankara UniversityORCID 0000-0002-6564-3133Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

Astrid R. Oudakker· 1 shared Britt MossinkiD· 1 shared Chantal SchoenmakeriD· 1 shared Dirk SchubertiD· 1 shared Filiz Çetinkaya· 1 shared Gözde VatanseveriD· 1 shared Hans van BokhoveniD· 1 shared Huiqing ZhouiD· 1 shared

Activity by Year

2019-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperNeuroscience

Astrid R. Oudakker

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2 papers

Recent·Most cited

Interval Timing Shows Selective Enhancement Under Psychosocial Stress: A Cortisol-Mediated Dissociation From Spatial Processing

Guvem Gumus Akay, Gözde Vatansever, Filiz Çetinkaya, Nuray Varol, Metehan Çíçek

This study examines how psychosocial stress affects temporal and spatial reproduction tasks, finding stress selectively enhances interval timing through cortisol-dopamine interactions.

bioRxiv · Nov 7, 2025 · PreprintNeuroscience

Neuronal network dysfunction in a human model for Kleefstra syndrome mediated by enhanced NMDAR signaling

Monica Frega, Katrin Linda, Jason M. Keller, Guvem Gumus Akay, Britt Mossink, Jon-Ruben van Rhijn, Moritz Negwer, Teun M. Klein Gunnewiek, Katharina Foreman, Nine F. Kompier, Chantal Schoenmaker, Willem M.R. van den Akker, Ilse van der Werf, Astrid R. Oudakker, Huiqing Zhou, , Nael Nadif Kasri

This study shows that EHMT1 deficiency in human neurons leads to NMDAR hyperfunction, causing neuronal network dysfunction in Kleefstra syndrome, and demonstrates pharmacological rescue of these phenotypes.

Nature Communications · Oct 30, 2019Peer-reviewedNeuroscience