Author

Kelly M. Grounds

University of Colorado BoulderORCID 0000-0002-6668-4818Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Michael H. B. StowelliD· 2 shared Addolorata PiscontiiD· 1 shared Charles A. HoefferiD· 1 shared Chong SheniD· 1 shared Daniel J. AdamsiD· 1 shared James Mapes· 1 shared Josien LevengaiD· 1 shared Kerri A. BalliD· 1 shared

Activity by Year

2017-2017 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersNeuroscience

Michael H. B. StowellORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintJun 15, 2017

Unexpected Early Proteomic Changes in Alzheimer’s Disease Model Mice Synaptosomes

Kerri A. Ball, Addolorata Pisconti, Kelly M. Grounds…·Michael H. B. Stowell

The paper uses proteomics to identify early changes in AD model mice synaptosomes, finding significant proteomic alterations before traditional disease markers appear.

1 citation

mouseproteomics

NeurosciencebioRxivPreprintApr 25, 2017

Synaptophysin is a β-Amyloid Target that Regulates Synaptic Plasticity and Seizure Susceptibility in an Alzhiemer’s Model

Daniel J. Adams, Chong Shen, Josien Levenga…·Michael H. B. Stowell

This study shows that Aβ42 binds to synaptophysin, disrupting its function and leading to impaired neurotransmission and increased seizure susceptibility in an Alzheimer’s model.

2 citations

mouseexperimental assay