Seralynne D. Vann

Electrophysiological properties of the medial mammillary bodies across the sleep-wake cycle

This study characterizes the electrophysiological properties of medial mammillary bodies across sleep-wake states in freely moving animals, identifying diverse firing patterns and their potential roles in memory processing.

Reduction in neurons immunoreactive for parvalbumin, calretinin and calbindin in the anteroventral thalamic nuclei of individuals with Down syndrome.

The paper examines changes in calcium-binding protein-expressing neurons in the anteroventral thalamic nuclei of individuals with Down syndrome compared to controls.

Reduced mammillary body volume in individuals with a schizophrenia diagnosis: an analysis of the COBRE data set

The paper analyzes mammillary body volume in schizophrenia patients using the COBRE dataset and finds reduced volume after controlling for brain size and gender.

Apolipoprotein ε4 modifies obesity-related atrophy in the hippocampal formation of cognitively healthy adults.

The paper examines how APOE ε4 modifies obesity-related atrophy in hippocampal subfields of cognitively healthy adults using neuroimaging data.

Stable encoding of visual cues in the mouse retrosplenial cortex

The paper shows that visual cue representations in the mouse retrosplenial cortex are stable and spatially selective, with stronger locomotion modulation than in primary visual cortex.

Mammillothalamic disconnection alters hippocampo-cortical oscillatory activity and microstructure: Implications for diencephalic amnesia

This study investigates how damage to the mammillothalamic tract affects hippocampo-cortical oscillatory activity and microstructure, linking these changes to memory impairments.

Striking reduction in neurons and glial cells in anterior thalamic nuclei of older patients with Down’s syndrome

The paper quantifies neuron and glial cell loss in anterior thalamic nuclei of older Down’s syndrome patients, finding a 70% neuron reduction and lesser glial loss, suggesting aging-related pathology in this region contributes to memory impairments.