Author

Bettina Halwachs

University of GrazORCID 0000-0002-7082-9346Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Systems Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 1 Research Focus:variant effect prediction· 1

Frequent Co-Authors

Sarah MasseriD· 2 shared Ulrich StelzliD· 2 shared Anna Katharina HolliD· 1 shared Anna Nusser· 1 shared Anton Emil Ludwig· 1 shared Elisabeth AnnereriD· 1 shared Evelyne Jany-LuigiD· 1 shared Gernot F. GrabneriD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersSystems Biology

Sarah MasserORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

The cytosolic PRMT5-CDK4 complex impairs cell cycle kinase signaling

Sarah Masser, Tanja Lotteritsch, Anton Emil Ludwig, Bettina Halwachs, Elisabeth Annerer, Marion Mußbacher, Ulrich Stelzl

The paper identifies PRMT5-CDK4 complex formation as a non-enzymatic regulator of cell cycle signaling, showing it modulates CDK4 interactions and mimics CDK4 inhibition.

bioRxiv · Dec 14, 2025 · PreprintSystems Biology

Mutational scanning pinpoints distinct binding sites of key ATGL regulators in lipolysis

Johanna M. Kohlmayr, Gernot F. Grabner, Anna Nusser, Anna Katharina Holl, Verina Manojlović, Bettina Halwachs, Sarah Masser, Evelyne Jany-Luig, Hanna Engelke, Robert Zimmermann, Ulrich Stelzl

The paper identifies switch mutations in ATGL that selectively disrupt interactions with specific regulatory proteins, linking these mutations to altered lipolysis activity and disease relevance.

Nature Communications · Mar 21, 2024Peer-reviewedSystems Biology