Author

Sonali Vishwa Mohan

Institute of BioinformaticsORCID 0000-0002-7159-7575Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:differential expression· 1 Organism:E. coli· 1 Assay:proteomics· 1

Frequent Co-Authors

Aparna Asok· 2 shared Harsha GowdaiD· 2 shared Kritika GuptaiD· 2 shared Pehu KohliiD· 2 shared Raghavan VaradarajaniD· 2 shared Shruti KhareiD· 2 shared Soumyanetra ChandraiD· 2 shared Arati RameshiD· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

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2 shared papersMolecular Biology

Aparna Asok

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2 papers

Recent·Most cited

The high mutational sensitivity of ccdA antitoxin is linked to codon optimality

Soumyanetra Chandra, Kritika Gupta, Shruti Khare, Pehu Kohli, Aparna Asok, Sonali Vishwa Mohan, Harsha Gowda, Raghavan Varadarajan

The paper shows that ccdA antitoxin in E. coli is highly sensitive to mutations, including synonymous ones, due to codon usage and operonic context effects on translation efficiency.

Molecular Biology and Evolution · Sep 5, 2022Peer-reviewedMolecular Biology

Loss of function from widely distributed, synonymous mutations at single codons

Kritika Gupta, Jyothi Prabha, Soumyanetra Chandra, Shruti Khare, Sonali Vishwa Mohan, Pehu Kohli, Aparna Asok, Harsha Gowda, Arati Ramesh, Raghavan Varadarajan

The paper shows that synonymous mutations can cause loss of function by altering RNA structure and gene expression ratios, validated in multiple operons.

bioRxiv · Jul 2, 2019 · PreprintMolecular Biology