Author

Renee C. Ryals

Oregon Health & Science UniversityORCID 0000-0002-7182-7238Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Allison G. CurtisiD· 1 shared Avi Weingarten· 1 shared Costas Andreas LyssiotisiD· 1 shared Emma Piraino· 1 shared Eric Weh· 1 shared Heather M. Hager· 1 shared Jocelyn F. KreyiD· 1 shared Katherine M. WehiD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperNeuroscience

Allison G. CurtisORCID

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2 papers

Recent·Most cited

NeuroscienceRedox BiologyPeer-reviewedJun 18, 2026

Redox imbalance dictates dependence on GOT1 versus GOT2 for rod photoreceptor health during aging and stress

Meini Chen, Eric Weh, Moloy T. Goswami…·Thomas J. Wubben

This study shows that GOT1 and GOT2 have opposing roles in rod photoreceptor health, with GOT2 acting as a neuroprotective factor under reductive stress conditions.

DOI 10.1016/j.redox.2026.104266

mouseexperimental assay

NeurosciencebioRxivPreprintFeb 28, 2023

Spontaneous allelic variant in Ush1g resulting in an expanded phenotype

Vladimir L. Vartanian, Jocelyn F. Krey, Paroma Chatterjee…·Peter G. Gillespie

The paper identifies a spontaneous mutation in the Ush1g gene in Neil1 knockout mice that causes an expanded phenotype including auditory and vestibular defects.

DOI 10.1101/2023.02.28.529432

mouseexperimental assay