Author

Kate Randall

Northwestern UniversityORCID 0000-0002-7199-9806Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Assay:electronic health records· 1 Research Focus:gene regulatory network inference· 1 Organism:rat· 1

Frequent Co-Authors

Amy LeeiD· 1 shared Angie HuangiD· 1 shared Boris V. ZemelmaniD· 1 shared Catherine W. MorgansiD· 1 shared Christof GaultiD· 1 shared Daiki FutagiiD· 1 shared Dayne Mayfield· 1 shared Gaoying Ren· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperNeuroscience

Amy LeeORCID

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2 papers

Recent·Most cited

The retinal light response is modulated by an mGluR5-mediated retrograde signal from ON-bipolar cells to photoreceptors

Catherine W. Morgans, Tammie L. Haley, Gaoying Ren, Ian A. M. Taylor, Kate Randall, Robert M. Duvoisin

This paper shows that mGluR5 in ON-bipolar cells modulates light response via endocannabinoid retrograde signaling, affecting ERG b-wave amplitude and intensity-response curves.

Journal of Neuroscience · Dec 10, 2025Peer-reviewedNeuroscience

A non-conducting role of the Cav1.4 Ca2+ channel drives homeostatic plasticity at the cone photoreceptor synapse

J. Wesley Maddox, Gregory J. Ordemann, Juan AM de la Rosa Vázquez, Angie Huang, Christof Gault, Serena R. Wisner, Kate Randall, Daiki Futagi, Nihal A. Salem, Dayne Mayfield, Boris V. Zemelman, Steven H. DeVries, Mrinalini Hoon, Amy Lee

The paper shows that a non-conducting Cav1.4 Ca2+ channel is essential for cone synaptogenesis, and its absence is compensated by Cav3 channels, leading to normal cone-mediated vision in knock-in mice.

eLife · Nov 12, 2024Peer-reviewedNeuroscience