Author

Kamran M. Ahmed

University of California, BerkeleyORCID 0000-0002-7204-5991Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2

Frequent Co-Authors

Helen S. BateupiD· 2 shared Polina KosilloiD· 2 shared Alexander H.C.W. Agopyan-MiuiD· 1 shared Bradley M. RobertsiD· 1 shared Corinna D. Wong· 1 shared Erin E. AisenbergiD· 1 shared Lisa ConyersiD· 1 shared Natalie M. DoigiD· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersNeuroscience

Helen S. BateupORCID

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2 papers

Recent·Most cited

Dopamine neuron morphology and output are differentially controlled by mTORC1 and mTORC2

Polina Kosillo, Kamran M. Ahmed, Erin E. Aisenberg, Vasiliki Karalis, Bradley M. Roberts, Stephanie Jane Cragg, Helen S. Bateup

This study investigates how mTORC1 and mTORC2 differentially regulate dopamine neuron morphology and function in mice, revealing distinct effects on structure, excitability, and dopamine output.

eLife · Jul 11, 2022Peer-reviewedNeuroscience

Tsc1-mTOR signaling controls the structure and function of midbrain dopamine neurons

Polina Kosillo, Natalie M. Doig, Kamran M. Ahmed, Alexander H.C.W. Agopyan-Miu, Corinna D. Wong, Lisa Conyers, Sarah Threlfell, Peter J. Magill, Helen S. Bateup

This study shows that loss of Tsc1 in midbrain dopamine neurons leads to structural and functional defects, causing cognitive flexibility deficits that can be mitigated by reducing Raptor.

Nature Communications · Nov 28, 2019Peer-reviewedNeuroscience