Author

Julie A. Rhoades Sterling

United States Department of Veterans AffairsORCID 0000-0002-7442-6085Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Organism:human· 1 Assay:imaging· 1 Approach:microscopy & imaging analysis· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Alexandra Gutierrez Vega· 1 shared Amber CrabtreeiD· 1 shared Andrea Gail MarshalliD· 1 shared Anishka Nagarimadugu· 1 shared Annet KiraboiD· 1 shared Antentor Othrell Hinton Jr.iD· 1 shared Arsh Jaiswal· 1 shared Audra LaneiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperSystems Biology

Alexandra Gutierrez Vega

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2 papers

Recent·Most cited

Three-dimensional electron microscopy uncovers mitochondrial network remodeling in residual triple negative breast cancer persisting after conventional chemotherapy treatments.

Mariah J. Berner, Heather K. Beasley, Benjamin Rodriguez, Audra Lane, Steven W. Wall, Andrea Gail Marshall, Zer Vue, Larry Vang, Mokryun L. Baek, Mason Killion, Faben Zeleke, Bryanna Shao, Dominique Parker, Autumn Peterson, Julie A. Rhoades Sterling, , Gloria Vittone Echeverria

This study uses 3D electron microscopy to analyze mitochondrial network changes in residual triple negative breast cancer after chemotherapy, revealing structural remodeling and lipid droplet interactions associated with chemoresistance.

iScience · Apr 24, 2026Peer-reviewedCancer Biology

A Computational Model of Tumor Interactions with Bone-Resident Cells Predicts Tumor-Type-Specific Responses to Perturbations

Alexandra Gutierrez Vega, Natalie E. Bennett, Erik P. Beadle, Saja Alshafeay, Ramcharan Chitturi, Anishka Nagarimadugu, Hrishikesh Villur, Arsh Jaiswal, Julie A. Rhoades Sterling, L. Lynn Harris

This paper develops a computational model of tumor-bone interactions to predict how different tumor types respond to perturbations, showing that bone-adapted tumors are less dependent on bone-derived growth factors and less responsive to OC-targeted therapy.

bioRxiv · Mar 19, 2026 · PreprintSystems Biology