Author

Camila Espasandín

Universidad de la República de UruguayORCID 0000-0002-7575-8970Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:mouse· 1

Frequent Co-Authors

Aldo J. CalliariiD· 1 shared Ana Paula MuletiD· 1 shared Camila Chiesa· 1 shared Carlos EscandeiD· 1 shared Geraldine SchlappiD· 1 shared José Luis BadanoiD· 1 shared José Manuel VerdesiD· 1 shared José Roberto Sotelo-SilveiraiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperPhysiology

Aldo J. CalliariORCID

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1 paper

Recent·Most cited

Adipocyte-specific deletion of Dbc1 does not recapitulate healthy obesity phenotype but suggests regulation of inflammation signaling

Leonardo Santos, Rafael Sebastián Fort, Geraldine Schlapp, Karina Cal, Valentina Pérez-Torrado, María Noel Meikle, Ana Paula Mulet, Camila Espasandín, Camila Chiesa, José Roberto Sotelo-Silveira, José Manuel Verdes, Paola Contreras, Aldo J. Calliari, Martina Crispo, José Luis Badano, Carlos Escande

This study shows that deleting Dbc1 in adipocytes does not cause healthy obesity but reveals a role for Dbc1 in inflammation during obesity.

PLoS ONE · May 2, 2025Peer-reviewedPhysiology