Author

Michael Marks

London School of Hygiene & Tropical MedicineORCID 0000-0002-7585-4743Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Michael Marks1 paper · University of Pittsburgh

Biology Disciplines

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Genetic & Genomic Medicine· 1 Genomics· 1

Signature Assays, Organisms & Methods

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Assay:whole-genome sequencing· 2 Approach:experimental assay· 1 Organism:human· 1 Research Focus:phylogenetics / evolution· 1 Approach:statistical model· 1 Research Focus:variant calling· 1

Frequent Co-Authors

Mathew A. BealeiD· 2 shared Nicholas Robert ThomsoniD· 2 shared Achyuta NoriiD· 1 shared Beauty Makamure· 1 shared Beth SampheriD· 1 shared Bianca Da Costa DiasiD· 1 shared Christina M. MarraiD· 1 shared Ethel DauyaiD· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersGenetic & Genomic Medicine

Mathew A. BealeORCID

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2 papers

Recent·Most cited

Phylo-Plex: A phylogenetically informed, low-cost amplicon sequencing platform for deployable high-resolution genomic epidemiology

Mathew A. Beale, Vignesh Shetty, Kirsty Ambridge, George Lacey, Sam Dougan, William Roberts-Sengier, Beth Sampher, Florent Lassalle, Matthew J. Dorman, Mahlape P. Mahlangu, Johanna M. E. Venter, Bianca Da Costa Dias, Martha Chipinduro, Tendai Washaya, Luanne Rodgers, , Nicholas Robert Thomson

Phylo-Plex is a low-cost amplicon sequencing method for high-resolution genomic epidemiology, achieving fine-scale lineage discrimination comparable to whole-genome sequencing in resource-limited settings.

Nature Communications · Jul 9, 2026Peer-reviewedGenetic & Genomic Medicine

Genomic epidemiology of syphilis reveals independent emergence of macrolide resistance across multiple circulating lineages

Mathew A. Beale, Michael Marks, Sharon K. Sahi, Lauren C. Tantalo, Achyuta Nori, Patrick French, Sheila A. Lukehart, Christina M. Marra, Nicholas Robert Thomson

The paper shows that macrolide resistance in syphilis emerged independently in multiple lineages, not just the SS14 lineage, challenging the idea that resistance drove its spread.

Nature Communications · Jul 22, 2019Peer-reviewedGenomics