Author

Glafira Ermakova

University of British ColumbiaORCID 0000-0002-7681-8007Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Genetics· 2 Neuroscience· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:C. elegans· 3 Approach:experimental assay· 2 Research Focus:differential expression· 1 Approach:statistical model· 1

Frequent Co-Authors

Junran YaniD· 3 shared Stefan TaubertiD· 3 shared Brendil Sabatino· 1 shared Catharine H. RankiniD· 1 shared Chiaki ShuzenjiiD· 1 shared Donna Seraj· 1 shared Forum BhanshaliiD· 1 shared Gahan DiwaniD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersGenetics

Junran YanORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

NeurosciencebioRxivPreprintAug 24, 2026

C. elegans Nuclear Hormone Receptor NHR-49 promotes attractive chemotaxis independently of its role in fatty acid metabolism

James Tao Wei Wu, Glafira Ermakova, Junran Yan…·Stefan Taubert

This study identifies a new role for NHR-49 in promoting attractive chemotaxis in C. elegans, independent of its known role in fatty acid metabolism.

DOI 10.64898/2026.08.19.745339

C. elegansexperimental assay

𝕏 PostBluesky

GeneticsNature CommunicationsPeer-reviewedFeb 18, 2025

Eukaryotic Elongation Factor 2 Kinase EFK-1/eEF2K promotes starvation resistance by preventing oxidative damage in C. elegans

Junran Yan, Forum Bhanshali, Chiaki Shuzenji…·Stefan Taubert

This study identifies efk-1/eEF2K as a key factor in C. elegans starvation survival by enhancing DNA repair and reducing oxidative stress through a translation-independent mechanism.

11 citations

C. elegansexperimental assay

𝕏 PostBluesky

GeneticsG3 Genes Genomes GeneticsPeer-reviewedJan 24, 2024

The unfolded protein response of the endoplasmic reticulum protects Caenorhabditis elegans against DNA damage caused by stalled replication forks

Jiaming Xu, Brendil Sabatino, Junran Yan…·Stefan Taubert

This study shows that depletion of DNA primases in C. elegans activates the endoplasmic reticulum unfolded protein response (UPR-ER), which is required to alleviate replication stress.

6 citations

C. elegansstatistical model

𝕏 PostBluesky