Junran Yan

This study identifies a new role for NHR-49 in promoting attractive chemotaxis in C. elegans, independent of its known role in fatty acid metabolism.

This study identifies efk-1/eEF2K as a key factor in C. elegans starvation survival by enhancing DNA repair and reducing oxidative stress through a translation-independent mechanism.

This study identifies Hoi1 as an adaptor that targets BLTP2-like proteins to ER-PM contact sites in yeast, regulating lipid homeostasis and showing conservation in worms and flies.

This study shows that depletion of DNA primases in C. elegans activates the endoplasmic reticulum unfolded protein response (UPR-ER), which is required to alleviate replication stress.

The paper shows that MDT-15 is essential for activating propionate shunt pathway genes in C. elegans, preventing embryonic lethality by regulating vitamin B12-dependent gene expression.