Author

Hannah Q. Estrada

Cedars-Sinai Medical CenterORCID 0000-0002-7905-2123Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:metagenomics· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Alka A. PotdariD· 1 shared Anna Y. Blackwood· 1 shared Dalton T. Stamps· 1 shared David Shih· 1 shared Dermot P.B. McGoverniD· 1 shared Emebet MengeshaiD· 1 shared Erica E. AlexeeviD· 1 shared Hussein HamadeiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperImmunology

Alka A. PotdarORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

TL1A overexpression in Crohn's Disease and mice alters Paneth cells and microbiota promoting ileal inflammation

Yuefang Ye, Shyam Kailashrao More, Hussein Hamade, Erica E. Alexeev, Yosuke Shimodaira, Anna Y. Blackwood, Dalton T. Stamps, Jordan H. Miller, Jay P. Abraham, Lisa S. Thomas, Sofi L. Castanon, Hannah Q. Estrada, Kotaro Kumagai, Alka A. Potdar, Talin Haritunians, , Kathrin S. Michelsen

This study shows that TL1A overexpression in Crohn's Disease and mice causes Paneth cell dysfunction and microbiota changes that promote ileal inflammation.

bioRxiv · Mar 2, 2025 · PreprintImmunology