Author

Kathrin S. Michelsen

Cedars-Sinai Medical CenterORCID 0000-0002-4974-6992Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:development & morphogenesis· 1 Assay:metagenomics· 1 Assay:proteomics· 1

Frequent Co-Authors

Aasritha NallapuiD· 1 shared Aayusha ThapaiD· 1 shared Alka A. PotdariD· 1 shared Anna Y. Blackwood· 1 shared Brian YangiD· 1 shared Christopher S. SeetiD· 1 shared Dalton T. Stamps· 1 shared David Shih· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperImmunology

Aasritha NallapuORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Mucosally sourced complement factor B modulates the host response to colitis

Aayusha Thapa, Aasritha Nallapu, Nnenna Muogboh, Swathi Nedunchezian, Khushi B Talati, Brian Yang, Jungheun Hyun, John Michael S Sanchez, Christopher S. Seet, Hon Wai Koon, Zhaoping Li, Matthew A. Ciorba, Kathrin S. Michelsen, Hrishikesh S. Kulkarni, Devesha Hrishikesh Kulkarni

This study shows that locally produced complement factor B in the gut reduces colitis, highlighting its role in mucosal immunity.

bioRxiv · Jan 1, 2026 · PreprintImmunology

TL1A overexpression in Crohn's Disease and mice alters Paneth cells and microbiota promoting ileal inflammation

Yuefang Ye, Shyam Kailashrao More, Hussein Hamade, Erica E. Alexeev, Yosuke Shimodaira, Anna Y. Blackwood, Dalton T. Stamps, Jordan H. Miller, Jay P. Abraham, Lisa S. Thomas, Sofi L. Castanon, Hannah Q. Estrada, Kotaro Kumagai, Alka A. Potdar, Talin Haritunians, , Kathrin S. Michelsen

This study shows that TL1A overexpression in Crohn's Disease and mice causes Paneth cell dysfunction and microbiota changes that promote ileal inflammation.

bioRxiv · Mar 2, 2025 · PreprintImmunology