Author

Max Keller

University of MichiganORCID 0000-0002-8095-8627Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1

Frequent Co-Authors

Albert Owusu GattoriD· 1 shared Annette Gabriele Beck‐SickingeriD· 1 shared Corinna SchüßiD· 1 shared David Jordan· 1 shared Fabian LießmanniD· 1 shared Jan SticheliD· 1 shared Jens MeileriD· 1 shared Liu LiuiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperBiochemistry

Albert Owusu GattorORCID

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2 papers

Recent·Most cited

V-ATPase-Driven Lysosomal Activation Orchestrates MEK2-Induced Endothelial Reprogramming

Ziad Sabry, Max Keller, Liu Liu, Morgan Salmon, Zhong Wang

This paper shows that MEK2 activation reprograms fibroblasts into endothelial-like cells by enhancing lysosomal activity through the V-ATPase-TFEB axis.

bioRxiv · Jun 10, 2026 · PreprintCell Biology

Identification of key Y4R residues enables the discovery of selective non-peptide small-molecule agonists

Tim Pelczyk, Corinna Schüß, Mateusz Sklodowski, Fabian Ließmann, David Jordan, Vivian Ehrlich, Paul Eisenhuth, Jan Stichel, Albert Owusu Gattor, Max Keller, Jens Meiler, Annette Gabriele Beck‐Sickinger

This study identifies key Y4R residues and discovers selective non-peptide small-molecule agonists through molecular docking, residue replacement, and ultra-large library screening.

bioRxiv · May 1, 2026 · PreprintBiochemistry